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Revision THA - workup, reconstruction and cages

History, radiographs, failed fixation, revision reconstruction and reinforcement cages

88 questions 7 source pages 1 images

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88 questions
Q1What is a Ganz reinforcement ring?📷▸
Ganz reinforcement ring
Ganz reinforcement ring
  • A reinforcement ring with a hook
  • Has screw holes for fixation
Q2How is a Ganz reinforcement ring fixed?▸
  • Bone graft on the defect
  • Fix the ring with screws
  • Then cement PE onto the ring
Q3Why does fixation of the Ganz ring depend only on screws?▸
  • There is no ingrowth or ongrowth onto the ring
  • Ring fixation depends only on the screws
Q4Describe the X-ray findings and what you would compare with.▸
  • Previous cemented THR with femoral lucency over Gruen zones 2, 3, 5, 6, 7, 8, 9, 11, 14 (8-14 anterior to posterior)
  • Multiple cortical lytic lesions with endosteal scalloping; no periosteal new bone; no soft tissue calcification
  • Compare previous X-ray for stem loosening: subsidence, implant migration, cement breakage, radiolucent line at bone-cement interface
Q5What are the risk factors for infection after THR?▸
  • Patient: elderly, immunocompromised - DM, CRF, steroids, alcoholism, recent infection
  • Disease: psoriasis, RA, prior hip operation, prior hip infection
  • Surgical: prolonged operation; post-operative complications UTI, skin necrosis, haematoma
Q6What should you look for in history and examination?▸
  • Immunocompromised conditions, cause of THR, early wound infection, repeated antibiotics, pain-free interval, preceding bacteraemia
  • Expect non-mechanical pain
  • PE: erythema, sinus
  • Classify using Tsukayama (positive intraop c/st, early postop, acute haematogenous, late chronic)
Q7What is the Tsukayama classification of total joint infection?▸
  • 1: positive intra-operative cultures
  • 2: early acute, <1 month post-op
  • 3: acute haematogenous (late acute)
  • 4: chronic
Q8What blood tests are used to investigate THR infection?▸
  • Leukocytosis, CRP >10mg/L, ESR >30; CRP sensitivity and specificity ~90%
  • IL-6 (precursor of CRP) is more accurate but may not be available
  • Berbari JBJS Am 2010: accuracy IL-6 > CRP > ESR > WBC
  • ESR 75%/70%, CRP 88%/74%, IL-6 97%/91% (sensitivity/specificity)
Q9How useful is hip aspiration and how is it performed?▸
  • Spangehl: sensitivity 0.86, specificity 0.94, PPV 0.67, NPV 0.98 (level 1); suggest in high clinical suspicion with abnormal ESR/CRP
  • Anterior approach: 1 inch lateral to femoral pulse, 1 inch below inguinal ligament; lateral approach at GT under XR guidance; US or XR guided
  • Off antibiotics for at least 1 week; no intra-articular LA (bacteriostatic)
  • Send immediately for GS/C+ST, PCR and total cell count (>3000 cells/ml, >80% polymorph); different instruments from 3 different sites, 2/3 samples positive
Q10What are the MSIS criteria and common pathogens for PJI?▸
  • MSIS: any one major or 3/5 minor
  • Acute <6 weeks: ESR not reliable, CRP >100, WBC >10000 cells/mL, neutrophil >90%
  • Chronic: ESR >30, CRP >10, WBC >3000 cells/mL, neutrophil >80%
  • Staph aureus and Staph epidermidis account for 2/3; also E. coli, enterococcus
Q11What are the principles of management of a chronic infected THR?▸
  • MDT approach with MSK radiologist, microbiologist, histopathologist, plastic surgeon and experienced revision arthroplasty surgeon
  • 2-stage revision: removal of implant, debridement and antibiotic-loaded cement spacer
  • Systemic antibiotics 6-12 weeks with monitoring; repeat hip aspiration at 6 weeks; CT to reassess bone stock
  • Second stage if infection free and serology negative x 2 weeks; must use cemented components (no ingrowth); frozen section negative if <5 polymorphs per 400x HPF
  • One stage only if host not immunocompromised, low-virulence organism, no sinus tract, no OM/loosening
  • 6 vs 12 weeks antibiotics non-inferior (NEJM 2021 Bernard RCT)
Q12How does the timing of infection determine management?▸
  • <4 weeks: acute infection with less biofilm - radical debridement, exchange of modular bearing (thorough debridement), IV antibiotics
  • >4 weeks: chronic infection, biofilm present - implant removal and antibiotic-loaded cement spacer
Q13How do the KLIC and CRIME 80 scores guide DAIR?▸
  • KLIC for Tsukayama type 2 early acute: kidney 2, liver 1.5, indication (fracture/revision) 1.5, cemented 2, CRP>115 2.5; score >4 = 55% failure rate
  • CRIME 80 for type 3 acute haematogenous: COPD 2, CRP>150 1, RA 3, indication fracture 3, male 1, exchange mobile component -1, age >80 2; score >3 = 60% failure
Q14What is the antibiotic-loaded cement recipe used for spacers?▸
  • Options are gentamicin, vancomycin, tobramycin
  • 1 pack cement (40g) with 1 bottle antibiotic (4g vancomycin); ~10% (~1.x g gentamicin)
  • No vacuum, no drain; local concentration falls to 1/2 after 10 days
Q15What are the reported success rates of one-stage and two-stage revision?▸
  • UCLH experience: TKR 1 stage 100%, 2 stage 93.2%
  • THR 1 stage 94.5%, 2 stage 91.8%
  • Ongoing ccc
Q16What history and examination findings are important in a painful THR?▸
  • History: age and premorbid state, nature of pain, infection symptoms, current function (HHS), details of last operation, rule out spine, neuro, PVD as causes of pain
  • PE: scar, deformity, sinus, ROM
  • ROM clues: stiff - HO/impingement/contracture; end range pain - impingement; all range pain - infection; IR/ER pain - femoral stem loose
  • Stinchfield test (resisted flexion ~SLR) suggests acetabular loosening; assess abductors
Q17What investigations are needed?▸
  • CT
  • Scannogram + scoli series
Q18What are the aims of revision THR?▸
  • Restore offset and LLD
  • Replenish bone stock
  • Secure and accurate placement of components
Q19What pre-operative preparation is required?▸
  • Counsel patient regarding prognosis of revision THR; check previous implant and approach
  • Prepare instruments for implant removal: AO large fragment, ex-plant, straight/curved osteotome, high speed burrs, cement splitters, US device, flexible light source, allograft, GTR plate and cables if planning ETO
  • Inform pathology lab for intraop frozen section; prepare cement spacer in case frozen section is positive
  • Prepare reconstruction options: long extended coated stem, jumbo cup/metal augment/cupcage/reinforcement rings, allograft
Q20What are the key intra-operative steps for femoral implant removal?▸
  • Intraop 1g tranexamic acid to reduce blood loss; old incision, protect sciatic nerve
  • Remove bone/cement at Gruen zone 1; try hammering implant out with anchorage at collar/head/hole
  • Remove cement mantle with osteotome, reamer, burr, K wires, cement splitter (split radially then longitudinally)
  • +/- trochanteric osteotomy/trochanteric page/ETO; if cement mantle is good, use tap in and tap out (cement on cement)
Q21What are the principles of an extended trochanteric osteotomy (ETO)?▸
  • Preserve abductor and vastus lateralis (blood supply to the flap); divide the lateralis insertion off the vastus tubercle and elevate vastus lateralis off the linea aspera
  • Less than 1/3 of the femoral circumference; length determined by the length of the cement mantle
  • Multiple drill holes then break with osteotome; fix with cables
Q22What is a bone window and why may implant removal be difficult?▸
  • Bone window by burr - create a trough over the posterior femur, remove the fenestrated part and dissect the bone block
  • Difficulty in removal of an AMA is due to bone growth into the fenestration
Q23How is a broken femoral stem removed?▸
  • From above: trephine reamer
  • From below: cortical window
  • From the side: ETO
Q24What ultrasonic and other methods are used to extract cement?▸
  • Fish-bone like ultrasonic tip - melts the cement, sinks into it, hardens when USG is off, then hammered out
  • Dish-like tip - just melts and lets cement leak through holes
  • Drill and tap; segmental cement extraction (long screw with multiple nuts, pour in new cement, hammer out each nut sequentially - cement is weak in tension)
  • Powered cement reamer - easy fracture especially over the lateral or anterior side
Q25How is the acetabular implant removed, including a ceramic liner?▸
  • Curved osteotome
  • Explant
  • Ceramic liner: disengage the smooth taperlock by a sharp tap into the liner, then use a suction cup with scallops that contact the peripheral rim of the metal shell to remove the liner
Q26How is reconstruction planned for the femur and acetabulum?▸
  • Prefer cementless components if previous cement was used - bone surface become sclerotic, making cement interdigitation difficult
  • Femur: long cementless stem with diaphyseal fixation and adequate distal scratch fit
  • Acetabulum: identify anatomy with landmarks, navigation, intraoperative imaging, robot
  • Rim defect: metal augment, strut graft; medial wall defect: morcelised bone graft, anti-protrusion cage
  • Pelvic discontinuity: cup cage construct; rim stable: line-to-line fit with screw fixation; standby dual mobility cup
  • Ideally 36mm head
Q27What are the principles and complications of femoral impaction bone grafting?▸
  • Retrograde canal filling with impacted particulate graft
  • Segmental defect -> contained defect -> allows subsidence + cement creep -> even load distribution -> bone remodelling
  • Technique: mesh + wiring creates a neomedullary canal for a cemented femoral stem; advantage: preserves bone stock
  • Complications: fracture during impaction, varus/valgus stem, incomplete cement mantle, subsidence (bone graft resorption)
Q28Describe the X-ray findings of this Charnley cemented total hip replacement.▸
  • Stem with small head, round back, collared, tapered on AP; cemented PE cup; fixation of trochanteric osteotomy
  • Femoral cement-bone interface radiolucency with absent cement at Gruen zones 5 and 6
  • Stem in varus with lateral cortical reaction (Harris: probable loosening)
  • >2mm radiolucency at DeLee and Charnley zones 1+2 (70% loosening)
  • Bone defect both sides: femur Paprosky type 2 with intact diaphyseal bone stock
  • Acetabulum likely AAOS type 3 - mixed segmental and cavitary defect
Q29How do you diagnose periprosthetic joint infection in this revision case?▸
  • Hip aspiration in 2 separate occasions for C/ST, cell count, alpha defensin
  • MSIS major criteria: sinus tract, +ve C/ST in 2 separate tissue/fluid
  • MSIS minor criteria: CRP/ESR, synovial WBC count, synovial neutrophil %, single +ve C/ST, >5 neutrophils in HPF
  • Chronic: ESR >30, CRP >10mg/L, synovial WBC >3000 cell/uL, PMN >80%
  • Acute: ESR not reliable, CRP >100mg/L, synovial WBC >10,000 cell/uL, PMN >90%
Q30If infection is excluded, what causes aseptic loosening and what is your pre-operative assessment?▸
  • Mild eccentric wear suggesting osteolysis
  • Request initial post-op X-ray: poor cementation technique (<2mm cement mantle) and poor implant positioning (varus stem, increased cup open angle) cause abnormal stress
  • Bloods for inflammatory markers and previous X-ray for comparison
  • Pre-op CT for bone stock, column and rim integrity; CT angiogram for proximity of great vessels to the cup
  • Trace old records for implant size, brand and removal instruments
  • If fit for OT, offer operation irrespective of symptoms to prevent further bone stock loss
Q31What are the goals, principles and key surgical considerations of revision hip surgery?▸
  • Goal: painless, stable, mobile hip free of infection
  • Principles: stable cup fixation with 70% host bone contact, replenish bone stock, restore hip centre, LLD and offset
  • Use previous approach, dissect from virgin plane, protect sciatic nerve and gluteus medius
  • Frozen section: >5 neutrophils/HPF means treat as infection
  • Long stem with extended coating for distal fixation; prepare allograft/ trabecular metal/ jumbo cup/ ring; accept high hip centre in the elderly
  • Prepare dual mobility cup and C/XLPE or M/XLPE articulation; slow post-op rehab due to higher dislocation rate
Q32How do you manage acetabular bone defects in revision surgery?▸
  • Cementless cup prerequisite: 70% cup coverage, 2/3 rim fit, intact columns
  • Paprosky: 1 minimal lysis; 2 <3cm superior migration; 3 >3cm superior migration with column not intact
  • Contained defect: avoid defect (high hip centre/ medialise), bone (autograft/allograft, morselized or structural, IBG), metal (wedge, jumbo/oblong cup, cage/ring), cement
  • Uncontained defect: convert to contained with mesh, then treat as contained with bone graft +/- augment ring
  • Impaction bone grafting: close segmental defects with flexible metal wire mesh, impact 8-10mm morselized trabecular graft, then cemented cup
  • Cages offload the graft by bridging ilium or ischium and a liner is cemented in; anti-protrusio cage for pelvic discontinuity
  • Jumbo cup >62mm women, >66mm men: simple with more host bone contact, but does not restore bone stock, may raise hip centre and has 10% dislocation rate
Q33What are the reasons for a broken cement mantle and how is an extended trochanteric osteotomy (ETO) performed?▸
  • Cement failure when stresses exceed the fatigue endurance limit of the stem-cement interface and the cement itself
  • Mantle must be homogeneous and even (2-5mm) with proximal medial cancellous bone <2mm
  • Stem must be smooth-contoured, not malaligned, with the tip filling >half of the distal medullary canal
  • ETO: drill holes at least 12cm from GT tip, segment is 1/3 of femoral circumference, hinged anterolaterally on periosteum/muscle
  • Apply cables distal to the osteotomy before reaming; fix with cerclage wires or cables
  • Indications: well-fixed stem, difficult cement removal, varus remodelling of proximal femur, need for enhanced acetabular exposure
Q34What is the aim of assessment when a total hip replacement is loose?▸
  • Rule out infection
  • Assess symptoms
Q35What history and examination findings are relevant in a loose total hip replacement?▸
  • Pain and constitutional symptoms
  • Surgical details: timing of OT, initial post-op infection
  • Recent infection, wound condition and any sinus
Q36What investigations are required before revision hip surgery?▸
  • Obtain previous X-ray for comparison
  • Blood test for inflammatory markers
  • Hip aspiration on 2 different occasions for C/ST, cell count and alpha defensin
  • Pre-op CT for bone stock, column and rim integrity
  • CT angiogram for proximity of great vessels to the acetabular cup
  • Trace old records for implant size, brand and removal instruments
Q37What are the MSIS 2011 major criteria for periprosthetic joint infection?▸
  • Sinus tract communicating with the joint
  • +ve C/ST in 2 separate tissue/fluid samples
Q38What are the MSIS 2011 minor criteria for periprosthetic joint infection?▸
  • CRP/ESR
  • Synovial WBC count and synovial neutrophil %
  • 1 C/ST +ve
  • 5 neutrophils in HPF
Q39What are the chronic periprosthetic infection thresholds in the MSIS score?▸
  • ESR >30
  • CRP >10mg/L
  • Synovial fluid WBC >3000 cell/uL
  • PMN >80%
Q40What are the acute periprosthetic infection thresholds in the MSIS score?▸
  • ESR not reliable
  • CRP >100mg/L
  • Synovial fluid WBC >10,000 cell/uL
  • PMN >90%
Q41What initial post-op X-ray findings suggest the aseptic loosening is technique-related?▸
  • Poor cementation technique with <2mm cement mantle
  • Poor positioning: varus stem position or increased open angle of the cup
  • These cause abnormal stress onto the implant
Q42What surgical approach and instruments should be prepared for revision hip surgery?▸
  • Use the previous approach
  • Prepare fibreoptic light source, XR, IM guide, osteotomes, ultrasonic cement removal device, cables, plates
Q43How are the sciatic nerve and gluteus medius protected during revision dissection?▸
  • Protect with hip extension and knee flexion
  • Stick to bone during dissection
  • Dissect from the virgin plane and extend the previous incision
Q44What is the role of frozen section during revision hip surgery?▸
  • Obtain intra-op samples before proceeding
  • If >5 neutrophils under high power view -> treat as infection
Q45When can a cementless acetabular cup be used in revision?▸
  • Achieve 70% coverage of the cup
  • 2/3 rim fit
  • Intact columns
Q46What is the AAOS classification of acetabular defects?▸
  • Types 1-5: segmental, cavitary, mixed, discontinuity, ankylosed
Q47Describe the Paprosky classification of acetabular defects.▸
  • Type 1: minimal lysis
  • Type 2: <3cm superior migration, column intact, rim distorted, >50% contact surface; cementless if >2/3 rim fit, otherwise augment ring
  • Type 3: >3cm superior migration, column not intact
  • X-ray features assessed: superior migration, tear drop, ilioischial line, ischial lysis
Q48How is a contained acetabular defect managed in revision hip surgery?▸
  • Avoid the defect: high hip centre or medialise
  • Bone: autograft/allograft, structural or morselized, impaction bone grafting
  • Metal: metal wedge, jumbo (60mm)/oblong cup, revision ring (Muller/Ganz) if host contact <70%, anti-protrusio cage for discontinuity, mesh for uncontained defect
  • Cement
Q49How is an uncontained acetabular defect managed?▸
  • Convert to a contained defect using mesh
  • Then treat as contained, usually with bone graft + augment ring
  • Use cemented fixation as there is no bone ingrowth from bone graft
Q50Which revision stem is chosen for a contained femoral defect?▸
  • Cemented stem is poorly fixed due to poor interdigitation with lost cancellous bone and sclerosis
  • Prefer extensively coated long stem in good bone stock
  • Severe bone loss with a large canal cavity is difficult - needs an extra large implant with risk of fracture and stress shielding (extra large implant for initial stability --> easy fracture during OT, significant stress shielding)
  • Alternative: impaction bone grafting + cemented stem
Q51How are segmental or cortical femoral defects managed in revision hip surgery?▸
  • Small segmental/cortical defect: cortical onlay strut graft (onlay struft graft)
  • Impaction bone grafting + mesh
  • Gross proximal bone defect: tumour prosthesis
Q52Describe the technique of impaction bone grafting in the acetabulum.▸
  • Close acetabular segmental defects with flexible metal wire mesh
  • Fill the contained defect with a layer of morselized 8-10mm trabecular graft, impacted tightly with hammer and impactors
  • Cement the cup
Q53What is the role of cages in managing acetabular bone defects?▸
  • Reconstruct the defect with allograft or trabecular metal
  • Offload the graft by bridging the defect, anchoring to the ilium or ischium
  • Liner cemented into the cage
Q54What are the advantages of a jumbo cup?▸
  • Defined as >62mm in women and >66mm in men
  • Relatively simple
  • Increased host bone-cup contact area maximising ingrowth
  • Minimises migration by force dissipation over a large area
  • Decreases the need for bone graft
Q55What are the disadvantages of a jumbo cup?▸
  • Bone stock is not restored
  • Limited applicability in oblong defect - may ream away the posterior column or wall
  • High dislocation rate due to acetabular size:femoral head ratio (10%) - may use dual mobility
  • May raise the hip centre of rotation
Q56How is a Paprosky type IIA (cavitary) acetabular defect reconstructed?▸
  • Hemispherical cup
Q57How is a Paprosky type IIB (segmental defect <30% rim) acetabular defect reconstructed?▸
  • Hemispherical cup or jumbo cup
Q58How is a Paprosky type IIC (protrusion) acetabular defect reconstructed?▸
  • Medial cancellous allograft
  • Hemispherical cup
  • Impaction bone grafting
Q59How is a Paprosky type IIIA (spherical remodelling, up and out) acetabular defect reconstructed?▸
  • Large hemispherical cup
Q60How is a Paprosky type IIIB (oblong remodelling) acetabular defect reconstructed?▸
  • Structural autograft/allograft with hemispherical cup
  • Trabecular metal shell with superior augment
  • High hip centre with hemispherical cup
Q61How is a Paprosky IIIB acetabular defect without discontinuity managed?▸
  • Non-biological: cancellous allograft with cage
  • Structural allograft (posterior column with cage)
  • Biological: trabecular metal with augments
  • Triflange custom implant
Q62How is an acute pelvic discontinuity (Paprosky IIIB) managed?▸
  • Compression
  • Plate with cage and allograft
  • Internal plate with trabecular metal
Q63How is a chronic pelvic discontinuity (Paprosky IIIB) managed?▸
  • Distraction
  • Acetabular transplant
  • Trabecular metal with augments
  • Triflange implant
Q64Describe Paprosky femoral defect type I.▸
  • Minimal metaphyseal bone loss
Q65Describe Paprosky femoral defect type II.▸
  • Extension of metaphyseal bone loss with an intact diaphysis
Q66Describe Paprosky femoral defect type IIIA.▸
  • Extensive metadiaphyseal bone loss
  • At least 4cm of intact cortical bone in the diaphysis
Q67Describe Paprosky femoral defect type IIIB.▸
  • Extensive metadiaphyseal bone loss
  • less than 4cm intact cortical bone in the diaphysis
Q68Describe Paprosky femoral defect type IV.▸
  • Extensive metadiaphyseal bone loss
  • Non-supportive diaphysis
Q69What cementless stem options are available for femoral revision?▸
  • Primary cementless stem
  • Diaphyseal fixation: extensively porous coated cylindrical stems
  • Wagner conical stem
  • Fluted tapered modular femoral stem
Q70What cemented stem options are available for femoral revision?▸
  • Primary cemented stem
  • Long cemented stem
  • Impaction bone grafting (IBG)
  • Cement-in-cement revision
Q71What other femoral reconstruction options exist for revision?▸
  • Allograft prosthetic composite
  • Tumour prosthesis
Q72What mechanical and biological risk factors predispose to failed hip fracture fixation?▸
  • Mechanical: fracture location and pattern, reduction quality, fixation method and quality
  • Biological patient factors: osteoporosis, smoking, compliance with rehab, reinjury
  • Fracture factors and infection
Q73What do you assess on history and examination of failed hip fracture fixation?▸
  • Pain, functional demands, fitness for surgery, patient expectations
  • Local: scars, sinus, sciatic nerve
  • Systemic: LLD, limb deformity, muscle power, walking status
  • XR: reason for failure, complications (AVN, arthritis, broken implant)
  • Scannogram, bloods +/- joint aspiration; CT for bone stock, GT healing, screw cut-out
Q74What are the management options for failed fracture fixation in hip fracture?▸
  • Joint salvage: revision fixation (unreliable fixation due to osteoporosis, defeats goal of early weight bearing)
  • Joint sacrifice: arthroplasty vs Girdlestone vs arthrodesis
  • Hemiarthroplasty vs THR
Q75Outline the intraoperative steps of revision surgery for failed hip fracture fixation.▸
  • Old incision; watch the sciatic nerve; release contracted tissue (glut max, anterior capsule, iliopsoas)
  • Remove implant after dislocation
  • Take frozen section samples to rule out infection before proceeding
  • Acetabulum: press-fit line to line, augment with screws, standby cemented cup; anatomical hip centre, medialise cup
  • Femur: cemented if fracture healed; cementless diaphyseal fixation bypassing fracture if not healed +/- calcar replacement/GT reattachment
  • 6. bearing articulation
Q76What are the targets for stability and rehabilitation after revision for failed fracture fixation?▸
  • Aim combined anteversion of 35 degrees
  • Standby dual mobility cup
  • +/- GT osteotomy to retension abductors
  • Rehab: hip spica, HO prophylaxis
  • Registry: no difference in 15-year cumulative survival between hybrid, cemented and cementless
Q77What pre-operative optimisation is required before revision for failed hip fracture fixation?▸
  • Preop anaesthetic and medical optimisation - revision OT is a long operation
  • Ulcer prophylaxis
  • DVT prophylaxis
Q78What are the general intra-operative considerations in revision for failed hip fracture fixation?▸
  • Osteopenic bone
  • Careful manipulation of the limb
Q79To what endpoint is the acetabulum reamed and why is the cup medialised in revision?▸
  • Ream to the point of exposing bleeding subchondral bone
  • Medialisation of the cup decreases joint reaction force (JRF)
Q80What are the indications for a reinforcement ring or cage in acetabular revision?▸
  • Rim defect preventing stable cementless cup fixation at the desirable position
  • Pelvic discontinuity
Q81What are the advantages of reinforcement rings and cages?▸
  • Immediate post-op: span/ bypass defect (span/bypass the defect), support bone graft so it can heal
  • Late post-op: incorporated bone graft confers long-term mechanical stability
Q82What does initial stability of a reinforcement ring or cage rely on?▸
  • Host bone factors: superior and inferior support
  • Superolateral defect - strut bone graft or augment
  • Fixation: supplementary screws
  • Decreases the seesaw effect
Q83What are the types of reinforcement rings and cages?▸
  • Containing: convert uncontained to contained defect (acetabular rim/medial wall mesh cage)
  • Lateralising: transfer load to roof and rim while decompressing bone graft (Muller ring)
  • Bridging: span major defect (Ganz ring, Burch-Schneider antiprotrusio cage)
  • All are used with a cemented PE cup
Q84Describe the Muller ring.▸
  • Provides anterior/medial wall support
  • Advantage: simple and easy
  • Disadvantage: cavitary defect only
Q85Describe the Ganz ring.▸
  • = Muller ring + inferior acetabular hook
  • Restores an appropriate centre of rotation (COR)
  • Decrease superior migration
Q86Describe the Burch-Schneider antiprotrusio cage.▸
  • Hemispherical shell with proximal flange + distal nose
  • Bridges the acetabular defect by anchorage at the ilium and ischium
Q87What are the indications for a Burch-Schneider cage?▸
  • Combined segmental + cavitary defect
  • Severe medial wall or posterior column defect
  • Pelvic discontinuity
Q88What are the disadvantages of a Burch-Schneider cage?▸
  • Large field needed
  • Lateralised hip centre
  • Technically demanding