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Foot and Ankle

Juvenile hallux valgus and physeal bracket

Juvenile hallux valgus management and physeal bracket deformity of the first ray

13 questions 3 source pages 1 images

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13 questions
Q1What is the pathophysiology of a physeal bracket?📷▸
Physeal bracket
Physeal bracket
  • Incomplete development of primary ossification centres
  • Aberrant physeal cartilage extends along the diaphysis and interferes with normal growth
  • Leads to a short, wide, triangular or trapezoidal phalanx
  • Longitudinally oriented cartilage bracket follows a C-shaped curve
  • Autosomal dominant when present in the hand, otherwise sporadic
Q2What conditions are associated with a physeal bracket?▸
  • Rubinstein-Taybi syndrome
  • Fibrodysplasia ossificans progressiva
  • Hallux varus in the feet
  • Clinodactyly and polydactyly in the hands
  • Apert's syndrome
Q3What is the radiographic staging?▸
  • Stage 1: delta-shaped diaphysis with radiolucent cartilaginous epiphysis
  • Stage 2: secondary ossification centres at proximal and distal ends
  • Stage 3: complete ossification of the bracket epiphysis along one side of the diaphysis
  • Stage 4: closure of the physis
  • MRI may be needed
Q4How is a physeal bracket managed?▸
  • Splinting to loosen soft tissues prior to surgery
  • Osteotomies (opening or closing wedge) in older children after closure of the bracket epiphysis
  • Physiolysis + interposition (PMMA or fat): earlier intervention allows more correction of angulation and more longitudinal growth
Q5What are the complications of nonoperative and operative treatment?▸
  • Nonoperative: structural deformation of the forefoot
  • Nonoperative: incomplete longitudinal growth of the affected bone
  • Nonoperative: clinodactyly at the interphalangeal joint
  • Operative: recurrence of deformity
  • Operative: incomplete correction of deformity
Q6How does juvenile hallux valgus differ from adult hallux valgus?▸
  • Usually large IMA, large DMAA +/- HVI, congruent MCPJ
  • Metatarsus primus varus
  • Ligamentous laxity with hypermobile 1st ray
  • Physis not yet closed
  • High recurrence
Q7What is the management of juvenile hallux valgus?▸
  • Address ligamentous laxity: TMT arthrodesis after physis closure for a hypermobile 1st ray
  • If physis open: osteotomy at medial cuneiform or wait until closure
  • Double osteotomy: medial cuneiform opening wedge + 1st MT osteotomy +/- Akin
  • Associated with flexible flatfoot and tight TA
Q8What is the management principle for RA foot deformity?▸
  • Relieve symptoms, preserve function, minimize complications
  • Goal: stable, pain-free plantigrade foot
  • Multidisciplinary: rheumatology, medical, anaesthetics, physio, OT
  • Conservative: physio, shoe wear (large toe box, rockerbottom), metatarsal pads or bars, NSAIDs
Q9Why does RA foot surgery favour fusion over reconstruction?▸
  • Primary disease is progressive, so reconstruction has a high chance of recurrence
  • need to bear in mind the risk of complications as a result of osteopenia, reduced vascularity, and immunosuppression
  • Look for other RA complications e.g. C1/2 subluxation, other lower limb arthritis
Q10What are the fusion positions and procedures used in the RA foot?▸
  • 1st MTPJ arthrodesis: 20 deg dorsiflexion, 10 deg valgus, neutral rotation
  • Lesser toes: principle is to decompress the joint to allow relocation, while maintaining the MT length cascade (Maestro's cascade)
  • MTPJ: Fowler (proximal P1 excision) or Stainsby (+ flexor-extensor interposition); IPJ: DuVries
  • Midfoot: TN +/- CC fusion; hindfoot: triple fusion
  • Ankle: synovectomy; fusion vs arthroplasty (JBJS 2007 systematic review, 70% satisfaction rate in both groups)
Q11What is the pathophysiology of RA foot deformities?▸
  • Stage I: synovitis
  • Stage II: joint erosion and tendon dysfunction
  • Stage III: progressive deformity
  • Forefoot: hallux valgus, claw toe, splay foot
  • Midfoot: rockerbottom + forefoot abduction; hindfoot: PTT insufficiency and disruption of the talocalcaneal interosseous ligament causing hindfoot valgus
Q12If the IMA is not severe but the HVA is severe, what does that suggest?▸
  • MCL rupture (usually acute, sesamoid not subluxed)
  • Metatarsus adductus
Q13How does the X-ray localize a flatfoot deformity?▸
  • X-ray localization - if at TMTJ, then it is a midfoot driven flatfoot. If at TNJ, is it s hindfoot driven flatfoot